![]() gambiae to the human malaria parasite Plasmodium falciparum, whereas Rel 1 activation reduced infection levels. Using RNA interference to deplete the negative regulators of these pathways, we found that Rel2 controls resistance of A. gambiae has been shown to be particularly critical to the mosquito's ability to manage infection with the rodent malaria parasite Plasmodium berghei. Rel1- and Rel2-dependent transcription in A. ![]() ![]() ![]() Immune responses mounted by the malaria vector Anopheles gambiae are largely regulated by the Toll and Imd (immune deficiency) pathways via the NF-kappaB transcription factors Rel1 and Rel2, which are controlled by the negative regulators Cactus and Caspar, respectively. ![]()
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